In their new study titled "LIS1 and NDEL1 Regulate Axonal Trafficking of Mitochondria in Mature Neurons", members of the Smith lab report a novel function of LIS1 and its protein partner NDEL1 in regulating mitochondrial dynamics in adult axons, raising the possibility that defects in the LIS1/NDEL1 dynein regulatory pathway could contribute to mitochondrial diseases with axonal pathologies.
Excerpt from UofSC's Biological Science Department post. Comments are closed.
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